Soj ntsuam duab

Kev kuaj mob tshwm sim ntawm tus kab mob tsuas yog tshwm sim tsis yog los ntawm hom mob ntshav qab zib mellitus, tab sis kuj los ntawm lub sijhawm ntawm nws cov chav kawm, cov neeg kawm tiav ntawm kev them nyiaj ntawm cov metabolism carbohydrate, muaj cov vascular mob hnyav thiab lwm yam kev cuam tshuam. Conventionally, cov tsos mob kho tau muab faib ua ob pawg:

cov tsos mobqhia tias decompensation ntawm tus kab mob,

cov tsos mob cuam tshuam nrog qhov loj thiab mob hnyav mob ntshav qab zib angiopathy,tshuaj mob hlwbthiab lwm tusua cov nyom nyuaj lossis nyuaj.

Qog Ntshavua rau muaj tsos ntawm glucosuria. Cov tsos mob ntawm ntshav qab zib (hyperglycemia):polyuria,polydipsia, poob phaus nrog ntau dua qab los noj mov, lub qhov ncauj qhuav, tsis muaj zog

microangiopathies (ntshav qab zib retinopathy,tshuaj mob hlwb,nephropathy),

macroangiopathies (atherosclerosishlab ntshav hlab plawv,aorta,GM hlab ntsha, qis qis dua), mob tshwm simntshav qab zib taw

pathology txuas ntxiv: furunculosis,colpitis,vaginitis, tso zis mob txeeb zig thiab lwm yam.

Kev kuaj mob

Hauv kev siv tshuaj, kev tsim nyog txaus rau kev kuaj mob ntawm hom 1 mob ntshav qab zib mellitus yog muaj cov tsos mob tshwm sim ntawm hyperglycemia (polyuria thiab polydipsia) thiab kuaj pom tseeb hyperglycemia - yoo capillary ntshav qab zib ntau dua 7.0 mmol / l thiab / lossis txhua lub sijhawm ntawm hnub ntau dua 11.1 mmol / l qhov chaw tsis teev 556 hnub

Thaum tsim kev kuaj mob, tus kws kho mob ua haujlwm raws li cov txheej txheem hauv qab no.

Tsis suav cov kab mob uas pom los ntawm cov tsos mob zoo sib xws (nqhis dej, polyuria, poob phaus): mob ntshav qab zib insipidus, psychogenic polydipsia, hyperparathyroidism, mob raum tsis ua haujlwm, thiab lwm yam. Theem no xaus nrog kev kuaj pom ntawm hyperglycemia syndrome.

Daim ntawv nosological ntawm ntshav qab zib tau teev. Ua ntej tshaj plaws, cov kab mob uas tau muaj nyob hauv pawg "Lwm hom mob ntshav qab zib" tsis suav nrog. Thiab tsuas yog tom qab no qhov teeb meem ntawm hom 1 ntshav qab zib lossis hom 2 mob ntshav qab zib tau raug daws tau. Kev txiav txim siab txog qib C-peptide ntawm lub plab khoob thiab tom qab ua exercise. Cov theem ntawm kev xav ntawm GAD tiv thaiv cov ntshav hauv cov ntshav kuj tau raug tshuaj xyuas.

Teeb meem

Qog ntshav qab zib tsis tsaug zog(Yog hais tias muaj ntau dhau ntawm cov tshuaj insulin)

Uas micro thiab loj heevtshuaj mob ntsws- kev ua txhaum ntawm permeabilitycov hlab ntsha, nce lawv cov tsis taus, nce zuj zus musmob ntsws vwmrau kev tsim khoatherosclerosisntshav hlab

Ntshav qab zib polyneuropathy—polyneuritislub dag zoghlab ntshamob raws cov hlab ntsha hauv pob tw,paresisthiabtuag tes tuag taw,

Ntshav qab zib mob pob txha- mob hauvcov pob qij txha, "Crunch", kev txwv ntawm kev txav mus los, txo qis hauv cov dej synovial thiab nce nws cov viscosity,

Ntshav qab zib ophthalmopathy- kev loj hlob thaum ntxovcataracts(clouding ntawm lub lens)hais qhia tawm(swbtshuaj retina),

Ntshav Qab Zib Nepropathy- Lub raum puas tsuaj nrog cov tsos ntawm cov protein thiab cov ntshav nyob hauv cov zis, thiab nyob rau hauv qhov mob loj nrog kev loj hlobglomerulonephritisthiablub raum tsis ua hauj lwm,

Ntshav qab zib txha caj qaum- kev hloov pauvpsychethiab lub siab lub ntsws, lub siab xav ua haujlwm lossiskev nyuaj siabcov tsos mob ntawm intoxicationCNS .

Kev Kho Mob Cov Ntsiab Cai

Lub hom phiaj tseem ceeb ntawm kev kho mob:

Kev tshem tawm ntawm txhua qhov kev soj ntsuam mob ntshav qab zib

Ua kom tau cov khoom noj khoom haus zoo tshaj plaws hauv kev tswj lub sijhawm.

Tiv thaiv tus mob ntshav qab zib thiab mob nyhav

Ua kom lub neej muaj txiaj ntsig zoo rau cov neeg mob.

Yuav kom ua tiav cov hom phiaj no:

dosed ib tug neeg lub cev ua si (DIF)

qhia cov neeg mob tswj tus kheej thiab cov kev yooj yim ntawm kev kho (tswj hwm lawv tus mob)