Pathological anatomy ntawm ntshav qab zib
Lub sij hawm ntev hyperglycemia pab txhawb kev tsim kho cov tshuaj tiv thaiv insulin thiab muaj kev cuam tshuam rau cov hlwb (qhov tshwm sim ntawm cov piam thaj hauv qab zib), ua rau cov ntshav qabzib transporter muaj protein ntau thiab p-cell secretory kev ua haujlwm. Tag nrho cov no txo qhov kev siv ntawm carbohydrates los ntawm cov nqaij thiab ua rau ua txhaum ntawm lwm hom metabolism. Raws li qhov tshwm sim, nyob rau hauv cov ntshav qab zib mellitus, qhov ua mob txhab rau ntau yam plab hnyuv siab raum thiab cov ntaub so ntswg tshwm sim. Cov neeg mob hloov loj heev tsis tsuas yog ua rau neeg mob leeg, tab sis kuj yog lub siab, cov hlab ntsha, retina, raum, thiab cov hlab ntsha hlwb (ntshav qab zib ua kom tsis muaj zog, retinopathy, nephropathy, neuropathy).
Kev txiav siab ntawm cov neeg mob uas tuag los ntawm ntshav qab zib yog txo qhov loj me, nrog hom I mob ntshav qab zib mellitus - qhov tsis sib xws vim muaj fibrosis, ua ke nrog cov lus hloov atrophic hauv lub lobules. Kev kuaj mob me me nthuav tawm me me me ntawm Langerhans nrog kev txo qis ntawm RASs. Hauv hom II mob ntshav qab zib mellitus, cov kab mob ua ke tuaj yeem muaj ntau ntxiv vim yog lipomatosis, tab sis cov lobules me me muaj nyob hauv ntu. Cov chav kawm ntawm ntshav qab zib mellitus ntawm ob hom yog txiav txim siab los ntawm mob ntshav qab zib angiopathies, yog li cov ntshav qab zib mellitus tseem hu ua tus kab mob metabolic vascular. Nws yog vim muaj kev puas tsuaj rau cov ntshav qab zib ua rau thawj qhov chaw ntawm qhov ua rau qhov muag tsis pom kev, hauv cov neeg mob raum no puas ntau dua 17 zaug, feem ntau 2-3 zaug ntau dua - myocardial infarction thiab strokes, 5 zaug ntau zaus - qis dua lim hiam ntau dua li cov neeg ntawm tib lub hnub nyoog thiab tub los ntxhais nrog normoglycemic ntsuas.
Kev mob ntshav qab zib macroangiopathy yog qhov txawv txav ntawm cov hlab ntsha ntawm cov nruab nrab thiab loj caliber thiab pom, raws li txoj cai, hauv cov neeg ntawm cov neeg laus thiab cov laus lub hnub nyoog, thiab yog li ntawd nws tau tshaj tawm nyob rau hauv hom II mob ntshav qab zib mellitus. Nws cov kev mob tshwm sim yog atherosclerosis, uas feem ntau tshaj tawm thiab ntau dua li cov tsis muaj ntshav qab zib (ntshav qab zib mellitus yog qhov muaj kev pheej hmoo rau atherosclerosis), thiab Menkeberg's mediacalcosis thiab diffuse intimal fibrosis muaj ntau dua. Raws li qhov tshwm sim ntawm yeej ntawm cov hlab ntsha loj, ntau necrosis thiab mob caj dab ntawm sab hauv qab loj hlob tuaj. Ntshav qab zib microcognition yog dav dav thiab loj hlob hauv cov neeg mob ntawm txhua lub hnub nyoog, thiab nws qhov cuam tshuam ncaj qha rau lub sijhawm mob ntshav qab zib mellitus tau sau tseg. Arterioles thiab capillaries ntawm ntau yam plab hnyuv siab raum thiab cov ntaub so ntswg raug cuam tshuam, tshwj xeeb tshaj yog feem ntau lub raum, retina, tawv nqaij thiab pob txha pob txha. Nrog rau cov kev hloov pauv tsis tseem ceeb (plasma soaking, vascular phab ntsa hyalinosis, plhaws, mob siab rau ntawm tes thiab atrophy), qhov tuab ntawm hauv qab ntawm daim nyias nyias ntawm cov leeg endothelial vim muaj kev sib txuam ntawm SHIK-cov tshuaj zoo (feem ntau hom IV collagen) kuj yog yam ntxwv ntawm ntshav qab zib mellitus.
Kev mob ntshav qab zib ua rau muaj ze li 100% ntawm cov neeg mob ntshav qab zib ntev dua 15 xyoos. Ntxiv nrog rau kev hloov pauv morphological ntawm tus kab mob ntshav qab zib microangiopathy uas underlies no qhov muag pathology, microaneurysms tsim nyob rau hauv cov hlab ntsha thiab cov leeg ntshav ntawm retina, thiab perivascular - edema, hemorrhages, dystrophic thiab atrophic hloov hauv qhov muag ntawm lub paj hlwb optic. Caws tej yam tsis muaj tawm, lossis yooj yim, cov mob ntshav qab zib retinopathy thiab loj hlob pauj.
1.3.2 Lub cev nqaij daim tawv
Sepsis nrog cov kab mob ntshiab yog hu ua septicopyemia. Feem ntau hom staphylococcal septicopyemia (fulminant thiab acute form).
1.3.2 Lub cev nqaij daim tawv
Sepsis nrog cov kab mob ntshiab yog hu ua septicopyemia. Feem ntau hom staphylococcal septicopyemia (fulminant thiab acute form).
Lub cev anatomy
Cov kev mob tshwm sim ntawm cov kab mob diverticular yog cov diverticula kom tsim nyog thiab cov kev hloov pauv hloov pauv ntawm phab ntsa plab hnyuv ua ntej lawv cov tsos ntawm cov nqaij thiab qib ntawm tes.
1.3.2 Lub cev nqaij daim tawv
Sepsis nrog cov kab mob ntshiab yog hu ua septicopyemia. Feem ntau hom staphylococcal septicopyemia (fulminant thiab acute form).
1.3.2 Lub cev nqaij daim tawv
Sepsis nrog cov kab mob ntshiab yog hu ua septicopyemia. Feem ntau hom staphylococcal septicopyemia (fulminant thiab acute form).
General pathology (lub cev anatomy thiab pathological anatomy)
Pathological anatomy (los ntawm Greek. Pathos - kab mob) - kev tshawb fawb uas tshawb pom cov qauv ntawm lub hauv paus pathological - sawv tawm nyob nruab nrab ntawm XVIII xyoo pua. Nws txoj kev loj hlob hauv cov keeb kwm niaj hnub raug xwm txheej raug faib ua ob ntu: macroscopic (kom txog rau thaum nruab nrab ntawm XIX caug xyoo.
TXAWV ANATOMY
Kev hloov pauv pathological nyob ntawm qhov teeb meem ntawm kev puas tsuaj rau cov hlab ntsha hlab ntsha los ntawm atherosclerosis. Nrog mob stenocardia, thaum tsis muaj myocardial infarction, tsuas yog foci me me ntawm cardiosclerosis tau sau tseg.
Pathological anatomy ntawm pulpitis
Hauv kev txheeb xyuas keeb kwm, mob ntsig txog cov kab mob ntawm cov leeg pib thaum pib (nrog mob txhaws mob ntawm cov leeg mob), yog los ntawm edema, hyperemia ntawm lub ntsws, nias ntawm cov hlab ntaws, uas yog ib qho ua rau mob hnyav.
3. Mob lub cev
Kev kis mob, nkag mus rau lub raum lossis lub plab mog los ntawm txoj kev hematogenous lossis urinogenous, ua rau cov nqaij mos ntawm lub raum thiab cov hlab ntsws ua haujlwm ntawm lub raum qhov ntswg.
Lub cev anatomy
Mob hlab pas es feem ntau txhim kho ntau dua hauv thaj chaw ib txwm muaj nqaim. Feem ntau, ib qho hlav tshwm sim hauv nruab nrab peb ntawm thoracic txoj hlab pas (40-70%). Cov hlav qes ntawm peb feem ntawm lub hauv siab yog nyob hauv thib ob qhov chaw hauv zaus (25-40%). Hauv sab thib peb ntawm lub hauv siab.