Acetylsalicylic Acid Hmoov

Txhawb pab lub enzyme MOR, cuam tshuam cov khoom tsim prostaglandin thiab ATP ntau lawm. Muaj tshuaj tiv thaiv thiab anti-inflammatory kev uainhibits kev sib sau ua ke platelet suav.

Qhov kev ua kom loog no yog vim ob qho tib si nruab nrab thiab ntu kev ua haujlwm. Nyob rau hauv cov ntaub ntawv ntawm febrile, nws txo qhov kub los ntawm kev ua ntawm qhov chaw thermoregulation.

Kev Sib Thuam thiab platelet nplaumli qub mob ntsws vwm txo qis vim yog lub peev xwm ntawm ASA los rhuav tshem qhov kev sib txuas ntawm thromboxane A2 (TXA 2) hauv platelets. Inhibits hluavtaws prothrombin (coagulation factor II ntshav) rau hauv daim siab thiab - nyob rau hauv ib koob tshuaj ntau tshaj 6 g / hnub. - nce PTV.

Cov Tshuaj Pharmacokinetics

Kev nqus cov tshuaj tom qab siv cov tshuaj hauv sab hauv yuav luag tiav. Lub sijhawm ib nrab ntawm kev tshem tawm ntawm cov tsis hloov pauv ASA tsis pub tshaj 20 feeb. TCmax ASK hauv ntshav ntshav - 10-20 feeb, tag nrho salicylate tshwm sim los ntawm metabolism hauv, - los ntawm 0.3 mus txog 2.0 teev.

Hauv kev cuam tshuam albumin lub xeev nyob rau hauv ntshav yog li 80% acetylsalicylic thiab salicylic acidsCov. Kev ua xyem xyav xyoob ntoo txawm tias lub cev nyob hauv daim ntawv protein.

Cov tshuaj txiav tawm hauv daim siab. Nws yog tawm los ntawm lub raum. Kev txiav tawm yog cuam tshuam los ntawm cov zis pH: thaum acidified, nws txo qis, thiab thaum alkalized, nws nce.

Cov tshuaj Pharmacokinetic ntsuas nyob ntawm seb qhov loj npaum li cas ntawm cov tshuaj npaum li cas. Kev tshem tawm cov khoom muag yog nonlinear. Ntxiv mus, hauv cov menyuam yaus ntawm 1 xyoo ntawm lub neej, hauv kev sib piv nrog cov neeg laus, nws ua tiav qeeb qeeb ntau dua.

Kev qhia rau kev siv: vim li cas cov tshuaj acetylsalicylic acid pab tau?

Kev qhia rau kev siv cov tshuaj acetylsalicylic acid yog:

• febrile kab mob nyob rau hauv kis thiab mob kab mob,
• rheumatoid mob caj dab,
• mob rwj,
• mob txhab myocardiumtshwm sim los ntawm cov tshuaj tiv thaiv immunopathological,
• kev hnov ​​mob mob ntawm ntau lub hauv paus pib suav nrog taub hau thiab mob hniav (nrog rau mob taub hau cuam tshuam nrog haus cawv haus cawv), mob pob txha thiab mob leeg, neuralgia, migraines,algomenorrhea.

Ib yam ntxiv tshuaj aspirin (los yog acetylsalicylic acid) yog siv los ua prophylactic yog tias raug teeb meem mob ntsws vwm,thromboembolism, IM (ntawm myocardial infarction cov tshuaj yog tawm rau kev tiv thaiv theem ob).

Cov Yuav Tsum Muaj

Nkag tau ASA yog contraindicated nyob rau hauv:

• Tshuaj aspirin hawb pob,
• lub sij hawm exacerbation erosive thiab ulcerative txhab ntawm lub kwj dej digestive,
• pais plab / plab hnyuv los ntshav,
• muaj vitamin K tsis txaus,
• hemophilia, hypoprothrombinemia, hemorrhagic diathesis,
• tsawg enzyme G6PD,
• portal tawg,
• raum / mob siab ua haujlwm
• aortic dissection
• thaum kho Methotrexate (yog tias ib zaug lub lis piam ib zaug ntawm cov tshuaj ntau dua 15 / mg),
• gouty mob caj dab, gout,
• cev xeeb tub (thawj peb thiab peb lub hlis yog meej contraindications),
• pub niam mis,
• muaj qhov tsis haum rau ASA / salicylates.

Sab sij huam

Kev phiv los ntawm kev kho mob ASA tuaj yeem tshwm sim nyob rau hauv daim ntawv ntawm:

• xeev siab
• mob ncauj plab,
• kev tsis nco qab
• kev fab tshuaj tsis haum
• zawv plab
• thrombocytopenia
• yaig thiab mob txhab tawm ntawm tus kwj dej digestive,
• lub raum thiab / lossis daim siab ua hauj lwm.

Nrog rau qhov siv ntev, tinnitus tshwm sim, hnov ​​pob ntseg poob qis, tsis pom kev tsis pom kev, kiv taub hau tshwm sim thiab, thaum noj ntau dua, mob taub hau. Los ntshav tseem ua tau. hypocoagulationntuav bronchospasm.

Acetylsalicylic acid, cov lus qhia rau kev siv (Txoj kev thiab ntau npaum li cas)

Ntawm nquag rheumatism cov neeg mob cov laus tau muab los ntawm 5 txog 8 g ntawm ASA ib hnub. Rau tus me nyuam, qhov tshuaj tau muab suav xam raws qhov hnyav. Raws li txoj cai, nws txawv ntawm 100 txog 125 mg / kg / hnub. Kev ntau ntau ntawm kev siv - 4-5 p. / Hnub.

1-2 lub lis piam tom qab pib qhov pib, qhov tshuaj rau tus menyuam raug txo mus rau 60-70 mg / kg / hnub, rau cov neeg mob laus, qhov tshuaj tsuas zoo li qub. Txuas kev kho mob txuas ntxiv yuav tsum ntev txog 6 lub lis piam.

Raws li cov lus qhia rau kev siv cov tshuaj acetylsalicylic acid, cov tshuaj yuav tsum tau txiav tawm maj mam dhau lub sijhawm 1-2 lub lis piam.

Acetylsalicylic acid rau mob taub hau thiab ua cov tshuaj kho rau qhov kub thiab txias yog tau hais tias noj tsawg dua. Yog li, nrog kev hnov ​​mob mob thiab febrile tej yam kev mob koob tshuaj rau 1 koob tshuaj rau ib tus neeg laus - los ntawm 0.25 txog 1 g nrog rau qhov tso cai ntawm daim ntawv thov los ntawm 4 txog 6 rubles ib hnub.

Nws yuav tsum nco ntsoov tias yog tias mob taub hau, ASA ua haujlwm tshwj xeeb yog tias qhov mob tshwm sim los ntawm qhov nce hauv ICP (mob siab intracranial).

Rau cov menyuam yaus, kev noj tshuaj zoo ntau ntawm lub sijhawm yog 10-15 mg / kg. Kev ntau ntau ntawm cov ntawv thov - 5 p. / Hnub.

Txoj kev kho yuav tsum tsis pub ntev tshaj 2 lub lis piam.

Txog ceeb toom mob ntsws vwm thiab embolism ASA noj 2-3 p. / Hnub. 0.5 g txhua. Los txhim kho rheological zog (rau liquefaction) ntshav cov tshuaj noj rau ntev npaum li cas ntawm 0.15-0.25 g / hnub.

Rau cov menyuam yaus hnub nyoog qis dua tsib xyoos, noj ib zaug yog 0.25 g, menyuam muaj plaub-xyoo tau tso cai muab 0.2 g ntawm ASA ib zaug, cov menyuam muaj ob xyoos - 0.1 g, ib-xyoo-0.05 g

Nws yog txwv tsis pub muab ASA rau menyuam yaus los ntawm qhov kub uas nce hauv qhov tom qab kis tus kab mobCov. Cov tshuaj ua rau tib lub hlwb thiab lub siab ua qauv qee cov kab mob, thiab ua ke nrog kis mob tuaj yeem ua rau menyuam loj hlobReye's syndrome.

Kev siv ntawm ASA hauv cosmetology

Acetylsalicylic acid lub ntsej muag lub ntsej muag tso cai rau koj kom tshem tawm sai sai, txo qhov nqaij o, tshem tawm liab, tshem cov txheej txheej saum npoo ntawm cov leeg tuag thiab ntxuav qhov hws ua paug.

Cov tshuaj ntws tawm ntawm daim tawv nqaij zoo thiab muaj tshuaj lom rau hauv cov rog, uas ua rau nws tsim nyog siv rau kev kho pob txuv: ntsiav tshuaj moistened nrog dej, thov rau cuam tshuam ntsiab ntawm lub ntsej muag los yog ntxiv rau qhov muaj pes tsawg leeg lub ntsej muag lub ntsej muag.

Acetylsalicylic acid los ntawm pob txuv ua haujlwm zoo hauv kev sib xyaw nrog txiv qaub kua txiv lossis zib ntab. Ua tau zoo rau kev daws teeb meem ntawm daim tawv nqaij thiab npog ntsej muag nrog av nplaum.

Txhawm rau npaj lub txiv qaub-tshuaj aspirin lub ntsej muag, ntsiav tshuaj (6 daim) tsuas yog hauv av nrog cov kua txiv tshiab kom txog thaum muaj ib homogeneous loj tau. Tom qab ntawv cov tshuaj muaj kiav rau khaus cov pob txuv thiab sab laug rau lawv kom txog thaum qhuav.

Daim npog ntsej muag nrog zib ntab yog npaj raws li hauv qab no: ntsiav tshuaj (3 daim) yog noo noo nrog dej, thiab tom qab ntawd, thaum lawv yaj tag, tov nrog 0.5-1 tablespoon (tshuaj yej) zib ntab.

Txhawm rau npaj daim npog av nplaum, 6 qhov ntxuaj ntawm ASA thiab 2 diav (me nyuam diav) ntawm av dawb / xiav av nplaum yuav tsum tov nrog dej sov.

Noj ntau dhau

Kev noj ntau dhau yuav tshwm sim los ntawm:

• kev kho mob ncua ntev ntawm ASA,
• ib qho kev tswj hwm ntawm qhov siab ntau dhau ntawm cov tshuaj.

Yog tus kos npe overdose salicylism syndrome, tshwm sim los ntawm dav dav malaise, hyperthermia, tinnitus, xeev siab, ntuav.

Yog tias muaj neeg coob dhau ntawm ASA, tus neeg raug tsim txom yuav tsum tau mus tom tsev kho mob sai. Nws lub plab ntxuav, lawv muab tshuab txais carbonkuaj CBS.

Ua raws li cov xwm txheej ntawm WWTP thiab kev sib npaug ntawm dej thiab electrolytes, kev qhia txog kev daws teeb meem yuav muab tshuaj sodium lactate, sodium citrate thiab sodium bicarbonate (raws li ib tug Txoj kev lis ntshav)

Yog tias pH tso zis yog 7.5-8.0, thiab cov ntshav plasma concentration ntawm salicylates ntau dua 300 mg / l (hauv menyuam yaus) thiab 500 mg / l (hauv cov neeg laus), yuav tsum tau saib xyuas mob hnyav alkaline diuretics.

Nrog rau intoxication loj hemodialysis, Ua rau lub ntsej muag ua kua dej, sau tshuaj kho mob.

Kev sib txuam

Txhim kho kev mob toxicity barbiturate npaj,valproic acid, methotrexatecov teebmeem ntawm qhov ncauj hypoglycemic tus neeg sawv cev, Digoxin, tshuaj yeeb qaug cawv, Triiodothyronine, tshuaj sulfa.

Tsis muaj zog cov teebmeem tshuaj kho mob (potassium-sparing thiab ntxau rov qab), antihypertensive tshuaj ACE inhibitorsuricosuric tus neeg sawv cev.

Nrog tib lub sijhawm siv nrog antithrombotic tshuaj, thrombolytics,cov tshuaj tiv thaiv tsis tau ntiag tug nce kev pheej hmoo uas los ntshav.

GCS txhim kho cov nyhuv ua kom tsis haum ntawm ASA rau ntawm qhov hnoos qeev ntawm lub plab zom mov, ua kom nws txoj kev tshem tawm thiab txo cov ntshav plasma.

Thaum siv ua ke nrog cov ntsev ntsev, Li nce ntxiv ntawm plasma concentration ntawm Li + ions.

Txhim kho lub tshuaj lom cawv ntawm cawv rau lub mucosa ntawm lub plab zom mov.

Cov lus qhia tshwj xeeb

Cov tshuaj yuav tsum tau siv nrog ceev faj hauv cov neeg nrog pathologies ntawm ob lub raum thiab mob siab, bronchial hawb pob, nrog rau cov ntshav nce ntxiv, decompensated lub plawv tsis ua haujlwm, thaum kho mob nrog anticoagulants, zoo li hauv cov neeg muaj keeb kwm ntawmerosive thiab ulcerative lesions ntawm lub plab zom mov thiab / lossis pais plab / plab hnyuv los ntshav.

Txawm hais tias nyob rau hauv cov koob tshuaj me me, ASA txo cov kev pom zoo. uric aciduas nyob rau hauv cov neeg mob raug tau yuav ua rau muaj mob hnyav mob gout.

Thaum noj cov tshuaj ASA ntau kawg lossis xav tau kev kho mob ntev ntev nrog tshuaj, nws yog ib qho tsim nyog yuav tsum soj ntsuam xyuas qib kom tsis tu ncua hemoglobin thiab tau ntsib kws kho mob.

Ua tus neeg sawv cev tiv thaiv kab mob, siv tshuaj ASA hauv ib koob li 5-8 g / hnub. txwv vim muaj ntau qhov kev pheej hmoo ntawm kev phiv tshuaj los ntawm cov hnyuv plab hnyuv.

Txhawm rau txo qis los ntshav thaum phais thiab tom qab lub sijhawm txiav, tom qab noj salicylates nres 5-7 hnub ua ntej kev phais mob.

Thaum noj tshuaj ASA, nws yuav tsum nco ntsoov tias qhov tshuaj no tuaj yeem noj tsis pub dhau 7 hnub yam tsis tau sab laj nrog kws kho mob. Ua tshuaj tua kabmob ASA, nws raug tso cai rau haus tsis pub ntau tshaj 3 hnub.

Tshuaj lom neeg lub zog ntawm cov tshuaj

Thaum ASA crystallizes, koob tsis muaj kob los yog monoclinic polyhedra nrog saj me ntsis qaub yog tsim. Cov muaju yog ruaj khov nyob rau hauv huab cua qhuav, tab sis nrog nce av noo, lawv maj mam hydrolyze rau salicylic thiab acetic acids.

Cov tshuaj nyob rau hauv nws daim ntawv dawb huv yog crystalline hmoov ntawm cov xim dawb thiab ua ntxhiab tsw. Cov tsos ntawm tsis hnov ​​tsw ntawm acetic acid yog qhov taw qhia tias cov tshuaj pib hydrolyze.

ASA raug rau esterification nyob rau hauv kev ua ntawm alkaline bicarbonates, alkaline hydroxides, zoo li hauv dej npau npau.

ASA tsis zoo rau hauv dej, soluble hauv chloroform thiab ether, yooj yim soluble hauv 96% ethanol. Kev daws teeb meem ntawm ASA hauv dej / aqueous media muaj ntau los cuam tshuam los ntawm pH qib: qhov ntau dua ntawm cov kuab tshuaj hnyav, cov tshuaj yooj yim yog yaj.

Tshuaj Acetylsalicylic acid-UBF, Asprovit, Aspinat, Aspivatrin, Nekstrim Ceev, Fluspirin, Taspir, Tshuaj aspirin

Hauv cov menyuam yaus, ASA siv tsis tau rau hyperthermiahais txog kis mob, vim tias qhov kev sib koom ua ke no tuaj yeem ua rau kev txhim kho kev mob tuag taus rau tus menyuam - Reye's syndrome.

Hauv cov menyuam mos yug tshiab, salicylic acid tuaj yeem hloov chaw vim albumin bilirubin thiab txhawb kev tsim kho txha caj qaum.

ASA nkag mus yooj yim rau txhua lub cev ntawm lub cev thiab cov nqaij, nrog rau cerebrospinal, synovial thiab peritoneal kua.

Nyob rau hauv qhov muaj edema thiab o, qhov nkag ntawm salicylate rau hauv kev sib koom ua ke tau nrawm. Hauv theem ntawm kev mob, ntawm qhov tsis sib xws, nws ua rau qeeb.

Dab tsi yog acetylsalicylic acid rau lub hangover?

ASA yog ib qho tshuaj zoo rau kev kho mob tsis zoo, vim tias muaj cov tshuaj antiplatelet zoo.

Txawm li cas los xij, nws yuav tsum nco ntsoov tias kev noj cov ntsiav tshuaj zoo dua tsis haus cawv, tab sis txog 2 teev ua ntej noj mov. Qhov no txo ​​txoj kev pheej hmoo ntawm kev kawm. microthrombi hauv cov hlab ntsha me me ntawm lub paj hlwb thiab - hauv ib feem - ntaub so ntswg edema.

Siv thaum cev xeeb tub thiab pub niam mis

Acetylsalicylic acid yog sib kis thaum cev xeeb tub. Tshwj xeeb hauv thawj peb thiab peb lub hlis ntawm kev ua kom tes taw. Hauv cov theem thaum ntxov, kev noj cov tshuaj tuaj yeem ua rau muaj kev pheej hmoo ntawm kev pheej hmoo tsis zoo thaum yug menyuam, hauv theem tom ntej - cuam tshuam kev xeeb tub thiab ua haujlwm tsis muaj zog.

ASA thiab nws cov metabolites hauv cov khoom me me txeem rau hauv cov mis. Tom qab kev tswj hwm kev siv tshuaj yaj yeeb, kev mob tshwm sim tsis pom hauv cov menyuam mos, yog li ntawd, raws li txoj cai, cuam tshuam ntawm kev pub niam mis (HB) tsis tsim nyog.

Yog tias tus poj niam pom tias kho mob ntev ntev uas muaj tus kab mob siab ASA ntau, nws yuav tsum tau tso tseg kom txhob muaj kab mob siab B.

Yuav luag txhua qhov kev tshuaj xyuas tshuav txog ASA tau zoo. Cov tshuaj yaj pheej pheej yig, ua tau zoo, muaj txiaj ntsig zoo, muaj kev siv ntau yam. Cov ntsiav tshuaj txig txo cov mob thiab kub ib ce, thiab kev noj tshuaj ASA tsis tu ncua hauv kev noj tshuaj me txo qhov kev pheej hmoo kev mob plawv nyob rau hauv cov neeg mob predisposed.

Qhov tsis zoo ntawm cov tshuaj feem ntau yog hu ua qhov tshwm sim phiv. Txawm li cas los xij, raws li neeg feem coob nco ntsoov, txhawm rau kom zam lawv, nws txaus kom ua raws li qee txoj cai thaum lub sijhawm kho: tsawg kawg, ua ntej noj tshuaj, koj yuav tsum ua tib zoo nyeem cov lus qhia thiab txiav haus cawv rau tag nrho lub sijhawm kho.

Pharmacological kev txiav txim

Acetylsalicylic acid yog cov tshuaj tsis-steroidal los tiv thaiv cov tshuaj uas muaj cov tshuaj antipyretic, analgesic, anti-inflammatory, antiaggregatory kev ua rau kev cuam tshuam ntawm kev ua si ntawm COX1 thiab COX2 uas tswj cov synthesis ntawm prostaglandins. Txhawb kev coj ua ke ntawm thromboxane A2 hauv platelets, txo kev sib sau, platelet adhesion thiab thrombosis. Tom qab kev tswj hwm hauv tsoomfwv ntawm kev daws teeb meem, cov nyhuv analgesic ntau dua li hais tawm tom qab qhov ncauj kev tswj hwm ntawm acetylsalicylic acid. Nrog subconjunctival thiab parabulbar tswj hwm, nws muaj suab tshaj tawm hauv zos los tiv thaiv kev cuam tshuam, uas pathogenetically qhia kev siv tshuaj rau kev kho mob ntawm cov txheej txheem hauv lub qhov muag ntawm ntau lub hauv paus pib thiab thaj chaw. Cov nyhuv tiv thaiv yog feem ntau tshaj tawm hais thaum siv cov tshuaj nyob rau hauv lub sij hawm mob ntawm cov txheej txheem inflammatory hauv lub qhov muag. Cov tshuaj txo cov tsos mob ua kom tsis haum khaus ntawm ib khub ntawm qhov muag sib deev.

Tsuas tshuaj thiab tswj hwm

Txhawm rau cov ntsiab lus ntawm ampoule (vial) nrog 25 mg lossis 50 mg ntawm cov tshuaj, ntxiv 2.5 ml lossis 5 ml dej rau txhaj tshuaj, ntsig txog, thiab co kom txog thaum yaj tag. Tsuas yog npaj tshiab 1% daws teeb meem ntawm acetylsalicylic acid siv.

Subconjunctival los yog parabulbar nyob rau hauv ib koob tshuaj tsis ntau tshaj 0.5 ml ntawm 1% kev daws teeb meem txhua hnub lossis txhua lwm hnub. Ib qho kev daws teeb meem 1% tuaj yeem siv rau hauv kev tsim qauv ntawm 12 tee txog 3-4 zaug hauv ib hnub.

Hauv kev kho cov txheej txheem mob hauv lub qhov muag, 1% tov yog siv rau hauv cov qauv ntawm instillations, 2 ncos 3-4 zaug hauv ib hnub.

Hauv kev kho cov kab mob endogenous thiab exogenous uveitis ntawm etiology, kho tshiab 1% daws yog muab subconjunctively hauv ib qho ntim ntawm 0.5 ml ib zaug ib hnub kom txog rau thaum cov txheej txheem inflammatory nres. Lub chav kawm ntawm kev kho mob yog 3-10 hnub. Ua raws li qhov mob hnyav ntawm cov txheej txheem inflammatory, subconjunctival tswj hwm tuaj yeem ua ke nrog instillations ntawm cov tshuaj 1-2 tee ntawm 1% kev daws txog li 5 zaug hauv ib hnub. Thaum muaj mob tshwm sim me me, ib tus tuaj yeem txwv tus kheej tsuas yog rau instillations ntawm 1-2 tee ntawm 1% kev daws 3-4 zaug ib hnub.

Kev tiv thaiv thiab kho mob ntawm cov mob uas tsis haum thiab tom qab muaj teeb meem 1% kev daws yog siv cov tshuaj subconjunctival lossis parabulbar hauv qhov ntim ntawm 0.3-0.5 ml ib hnub ib zaug. Lub chav kawm ntawm kev kho mob yog 9-10 hno.

Kev tiv thaiv kab mob macular edema tom qab kev ua haujlwm ntsig txog kev tshem tawm cataract thiab kev cog hniav ntawm lub ntsej muag cuav, cov tshuaj npaj tau siv nyob rau hauv cov qauv ntawm cov tshuaj 1%, 1-2 ncos 3-4 zaug hauv ib hnub rau 4 lub lis piam tom qab tshem tawm cataract.

Sab sij huam

Nrog rau siv tshuaj yeeb nyob hauv cov tshuaj pom zoo ntau npaum li cas, cov tshuaj tiv thaiv rau lub cev tsis muaj kev sib haum xeeb.

Nrog subconjunctival tswj hwm, qhov pom ntawm chemosis yog ua tau, uas daws tau hauv ob peb teev. Kev mob siab thiab mob tas mus li hauv qhov chaw txhaj tshuaj yog cov mob me, qhov ncua sij hawm ntawm qhov tsis hnov ​​zoo yog 5-7 feeb. Txhawm rau tiv thaiv mob thaum lub sijhawm ua haujlwm subconjunctival lossis parabulbar tswj hwm, nws raug tso cai siv 2% tshuaj tiv thaiv procaine ua cov kuab tshuaj rau hauv kev npaj ntawm acetylsalicylic acid kua.

Qee zaum hauv thaj chaw txhaj tshuaj, yuav muaj cov ntaub so ntswg, subconjunctival hemorrhage, uas tshem tawm los ntawm kev siv 3% poov tshuaj iodide nyob rau hauv daim ntawv ntawm instillations 4-5 zaug hauv ib hnub.

Kev muaj peev xwm tsim kho ntawm xeev siab, txo qis kev qab los noj mov, raws plab, qhov txhab tshwm thiab mob tawm hauv cov hnyuv, ua xua (tawv nqaij ua pob ua xua, mob caj dab), mob siab thiab / lossis lub raum tsis ua haujlwm, thrombocytopenia, bronchospasm.

Daim ntawv thov nta

Npaj ua tsim yuav tsum tau siv thaum nruab hnub. Tsis txhob sib tov cov tshuaj txhaj ntawm cov tshuaj nrog cov kev daws ntawm lwm cov tshuaj uas tsis muaj npe hauv cov lus qhia no. Cov tshuaj ua tau zoo sib xws nrog procaine (hauv ib qho syringe). Yog tias nws yog qhov yuav tsum tau muab tshuaj rau acetylsalicylic acid ib txhij nrog lwm cov tshuaj rau etiotropic thiab / lossis kev kho mob, tsawg kawg 10-15 feeb yuav tsum elapse ntawm kev siv ntau yam tshuaj ophthalmic. Lub chav kawm ntawm kev kho mob yuav tsum tsis pub tshaj 10-12 hnub. Thaum kho, cov iav tsom qhov muag yuav tsum tsis txhob hnav.

Txog kev tiv thaiv kab mob postoperative hemorrhagic (tshwj xeeb tshaj yog rau cov neeg mob ntshav qab zib mellitus), kev siv tshuaj ua ntej ntawm angioprotectors (dicinone, etamsylate, thiab lwm yam) tau pom zoo.

Kev tiv thaiv kev nyab xeeb

Kev siv cov tshuaj yuav tsum tau ceev faj thaum muaj kev cuam tshuam ntawm cov ntshav coagulation system thiab cov kab mob ntawm txoj hnyuv thiab ua haujlwm ntawm txoj hnyuv hauv lub pob txha mus rau qhov pom ntawm kev muaj peev xwm los ntshav. Nrog perforated qhov txhab ntawm qhov muag nrog kev puas tsuaj rau lub cev ciliary, hemorrhage yog tau.

Acetylsalicylic acid txawm nyob rau hauv cov koob tshuaj me me txo qhov kev tsis haum ntawm uric acid los ntawm lub cev, uas tuaj yeem ua rau muaj kev txhim kho ntawm kev mob hnyav ntawm gout nyob rau hauv cov neeg mob yooj yim. Thaum kho, ib qho yuav tsum tsis kam noj ethanol.

Cuam tshuam rau kev muaj peev xwm tsav tsheb thiab tswj cov cuab yeej muaj peev xwm txaus ntshai: cov neeg mob uas ua rau lawv qhov tsis pom kev zoo tom qab siv lub qhov muag tsis pom zoo kom tsav tsheb los yog ua haujlwm nrog txav cov txheej txheem rau ob peb feeb tom qab kev siv tshuaj.

Lub Npe Tsis Yog Neeg Tsis Muaj Npe

INN: Acetylsalicylic acid.

Cov tshuaj aspirin hmoov yog txoj kev kho mob zoo rau txo cov tsos mob ntawm tus mob khaub thuas thiab mob khaub thuas.

Cov hmoov nyob rau hauv cov muaj pes tsawg leeg muaj ntau lub tebchaw ib zaug. Ntawm lawv: acetylsalicylic acid 500 mg, chlorpheniramine thiab phenylephrine. Cov khoom siv ntxiv yog: sodium bicarbonate, ib qho me me ntawm citric acid, kua qaub tsw thiab xim daj.

Hmoov nyob rau hauv daim ntawv ntawm cov me me. Yuav luag txhua lub sijhawm muaj xim dawb, qee zaum nrog lub zas daj. Effervescent hmoov yog npaj rau kev npaj ntawm kev daws. Ntim rau hauv ib lub hnab ntawv tshwj xeeb laminated.

Effervescent hmoov yog npaj rau kev npaj ntawm kev daws.

Dab tsi pab Aspirin hmoov

Aspirin Complex (tshuaj aspirin complex) yog siv los ua ib qho ntawm cov tsos mob rau tshem tawm mob thiab mob khaub thuas. Nws cov nyhuv yog qhov ncaj ncees vim yog ua rau ntawm cov txuam nrog cov khoom xyaw uas muaj nyob hauv cov hmoov.

Cov cim tseem ceeb rau kev siv:

• kev kho mob mob hniav thiab mob taub hau,
• myalgia thiab arthralgia,
• mob caj pas
• cov kev kho txoj hauv kev kho mob rau cov mob ua pa sab saud,
• coj khaub ncaws
• mob nraub qaum
• kub taub hau thiab kub taub hau, ua kom pom tus mob khaub thuas thiab lwm yam kab mob sib kis ntawm qhov mob tshwm sim.

Cov kev ntsuas no yog npaj rau cov neeg laus thiab cov menyuam hnub nyoog dhau 15 xyoos. Tab sis qhov ntau npaum li cas thiab lub sijhawm kho yog txiav txim siab ib tus zuj zus rau txhua tus neeg mob, raws qhov mob hnyav ntawm cov tsos mob tshwm sim hauv chaw kuaj mob.