Siv cov piam thaj hauv cov ntsiav tshuaj kom raug

Cov tshuaj muaj nyob rau hauv daim ntawv ntawm cov ntsiav tshuaj thiab ib qho kev daws teeb meem rau kev tswj kav. Lub hauv paus ua hauj lwm tseem ceeb ntawm Glucose yog dextrose monohydrate, nws cov ntsiab lus yog hauv:

• 500 mg rau ib ntsiav tshuaj
• 100 ml ntawm kev daws - 40, 20, 10 thiab 5 g.

Qhov muaj pes tsawg leeg ntawm cov xaim xyaw ua ke ntawm kev daws suav nrog dej rau txhaj tshuaj thiab hydrochloric acid.

Cov tshuaj nkag rau hauv lub khw muag tshuaj network:

• Ntsiav tshuaj - hauv cov hlwv pob ntawm 10 daim,
• Kev daws rau Txoj kev lis ntshav - hauv cov thawv yas ntawm 50, 100, 150, 250, 500, 1000 ml lossis hauv cov iav fwj ntawm 100, 200, 400, 500 ml,
• Kev daws rau kev tswj hwm kev ncaj ncees yog hauv 5 ml thiab 10 ml iav ampoules.

Kev ntsuas rau siv

Raws li cov lus qhia rau Glucose, cov tshuaj siv yog siv los them rau cov tsis muaj peev xwm txog cov carbohydrates hauv lub cev uas tshwm sim tawm tsam keeb kwm ntawm ntau pathologies.

Cov kua nplaum no tseem koom nrog rau kev kho mob nyuaj rau:

• Kev kho lub cev qhuav dej uas tshwm sim tom qab lub sijhawm dhau los lossis los ntawm ntuav thiab zawv plab,
• Lub cev intoxication,
• Daim siab ua rau lub siab, mob siab, mob ntshav siab thiab mob ntshav hauv lub siab,
• Hemorrhagic diathesis,
• Kev Ntshav Qab Zib,
• Poob siab thiab vau.

Cov Yuav Tsum Muaj

Kev siv cov kua nplaum nyob hauv daim ntawv ntawm txoj kev daws teeb meem yog contraindicated rau hauv cov neeg mob uas muaj keeb kwm muaj cov kev ua haujlwm thiab cov kab mob:

• Decompensated mob ntshav qab zib mellitus,
• Qog Ntshav,
• Hyperlactacidemia,
• Kev mob tsis zoo ntawm qabzib siv,
• Hyperosmolar coma.

Nrog kev ceev faj, kev tswj hwm cov tshuaj ntawm cov tshuaj yog raug rau cov neeg mob nrog:

• Decompensated mob plawv tsis ua haujlwm,
• Kev Hyponatremia,
• Mob raum tsis ua hauj lwm.

Ib qho ntxiv, cov ntsiav tshuaj ntshav qab zib yuav tsum tsis txhob noj nrog:

• Mob ntshav qab zib mellitus

• Circulatory pathologies, nyob rau hauv uas muaj qib siab ntawm kev pheej hmoo ntawm lub ntsws lossis hlab hlwb o,
• Mob laug ventricular tsis ua hauj lwm,
• O o rau lub hlwb los yog lub ntsws
• Tshaj dhau.

Tsuas tshuaj thiab tswj hwm

Cov kua nplaum (glucose) noj tau qhov ncauj 1.5 teev ua ntej noj mov. Kev noj tshuaj ib zaug yuav tsum tsis pub ntau tshaj 300 mg ntawm qhov tshuaj ib 1 kg ntawm tus neeg mob qhov hnyav, noj hauv ib teev.

Cov kua nplaum nyob rau hauv cov kua dej los ntawm txoj kev nrog kua los yog dav hlau txoj kev, qhov teem caij tau tsim los ntawm tus kws kho mob uas koom nrog.

Raws li cov lus qhia, qhov ntau tshaj txhua hnub tshuaj rau cov neeg laus nrog Txoj kev lis ntshav yog rau:

• 5% isotonic dextrose kev daws teeb meem - 2000 ml, kev tswj hwm ntawm 150 tee ib feeb lossis 400 ml ib teev,
• 0% hypertonic daws - 1000 ml, nrog qhov ceev ntawm 60 tee ib feeb,
• 20% kev daws - 300 ml, ceev - txog 40 ncos ib feeb,
• Kev daws 40% - 250 ml, qhov txhaj tshuaj siab tshaj plaws yog nce mus txog 30 tee hauv ib feeb.

Thaum muab tshuaj rau ntshav qab zib rau menyuam yaus, kev muab tshuaj yog tsim raws qhov hnyav ntawm lub cev ntawm tus menyuam, thiab yuav tsum tsis txhob ntau tshaj cov ntsuas hauv qab no:

• Nrog rau tus menyuam yaus qhov hnyav ntawm 0 txog 10 kg - 100 ml rau 1 kg ntawm qhov hnyav hauv ib hnub,
• Cov menyuam ntawm 10 mus rau 20 kg - 50 ml rau txhua kg dua 10 kg hauv ib hnub yog ntxiv rau 1000 ml
• Rau cov neeg mob hnyav dua 20 kg - txog 1500 ml ntxiv 20 ml ib kg ntau tshaj 20 kg ib hnub.

Kev tswj xyuas dav hlau ntawm 5% thiab 10% kev daws teeb meem yog kho nrog ib zaug tshuaj ntawm 10-50 ml.

Nyob rau hauv rooj plaub thaum Glucose ua cov tshuaj tseem ceeb rau kev tswj hwm kev coj noj coj ua ntawm lwm cov tshuaj, qhov nyiaj ntawm cov tshuaj tau daws rau hauv ib qho ntim ntawm 50 txog 250 ml ib koob ntawm cov tshuaj. Tus nqi ntawm kev tswj hwm hauv qhov no yog txiav txim siab los ntawm cov yam ntxwv ntawm cov tshuaj yaj hauv nws.

Sab sij huam

Raws li cov lus qhia, Glucose tsis ua kev phem rau lub cev nrog kev teem caij kom raug thiab ua raws li cov cai ntawm kev siv.

Cov kev mob tshwm sim los ntawm cov tshuaj muaj xws li:

• Mob laug ventricular tsis ua hauj lwm,
• Qog Ntshav,
• Hypervolemia
• Polyuria
• Ua npaws.

Tej zaum qhov tshwm sim ntawm qhov mob ntawm thaj chaw ntawm kev tswj hwm, kev hloov hauv zos hauv daim ntawv ntawm doog, thrombophlebitis, kev txhim kho ntawm kev kis mob.

Cov lus qhia tshwj xeeb

Kev siv cov kua nplaum nyob hauv lub sijhawm cev xeeb tub thiab pub niam mis.

Rau cov neeg mob ntshav qab zib, cov tshuaj tau tswj hwm nyob rau hauv kev tswj hwm ntawm cov piam thaj hauv cov ntshav thiab zis.

Kev ua txhaum yog nqa tawm hauv tsev kho mob, hauv kev ua raws li tag nrho cov cai asepsis.

Thaum ua ke nrog lwm cov tshuaj, qhov sib xyaw ntawm cov tshuaj yog tswj tau pom, qhov sib xyaw ua ke yuav tsum yog pob tshab yam tsis pom kev ncua. Nws yog ib qho tsim nyog yuav tsum tau npaj cov khoom qab zib nrog cov kua nplaum sai sai ua ntej kev tswj hwm tus txheej txheem; nws yog txwv tsis pub siv qhov sib xyaw txawm tias tom qab kev khaws cia luv.

Kev npaj nrog tib cov tshuaj muaj zog: Glucosteril, Glucose-Eskom, Dextrose-Vial thiab lwm yam.

Glucose analogs, tshuaj zoo sib xws hauv cov haujlwm ntawm kev ua: Aminoven, Hepasol, Hydramine, Fibrinosol thiab lwm yam.

Yeeb tshuaj sib cuam tshuam

Siv tib lub sijhawm dhau los ntawm catecholamines thiab tshuaj steroids txo cov piam thaj kev khaws cia.

Nws tsis yog qhov tsis suav hais tias muaj kev cuam tshuam ntawm cov dej-electrolyte sib npaug ntawm dextrose cov kev daws teeb meem thiab cov tsos ntawm cov nyhuv glycemic thaum siv ua ke nrog cov tshuaj uas cuam tshuam rau cov dej-electrolyte tshuav thiab muaj cov nyhuv hypoglycemic.

Cov tshuaj tua ntshav (analogues) yog: kev daws teeb meem - Glucosteryl, Glucose Bufus, Glucose-Eskom.

Kev taw qhia thiab contraindications rau siv

Cov kua nplaum tsim nyob rau hauv cov ntsiav tshuaj yog tawm rau:

• carbohydrate tsis zoo
• hypoglycemia (cov piam thaj hauv ntshav tsawg),
• kev lom nrog tshuaj lom neeg mob hepatotropic (paracetamol, aniline, carbon tetrachloride) ntawm kev ua kom me me mus rau mob ib nrab,
• lub cev qhuav dej (raws plab, ntuav).

Qhov kev tiv thaiv tsis zoo rau kev siv cov tshuaj no yog qhov muaj hyperglycemia (ntshav qab zib ntshav siab) hauv tus neeg mob, ntshav qab zib mellitus, hyperlactacidemia, hyperhydration thiab mob laug ventricular tsis ua haujlwm. Tsis txhob siv dextrose nrog hypersmolar coma, nrog o ntawm lub hlwb thiab / lossis mob ntsws.

Kev siv tshuaj ntev ntev tuaj yeem pab txhawb kev txhim kho hypokalemia (nyob rau hauv cov ntshav, cov concentration ntawm cov potassium ions poob qis), hypervolemia (nce ntim ntawm circulating plasma thiab ntshav) thiab hyperglycemia.

Cov yam ntxwv ntawm kev siv kua nplaum

Dextrose ntsiav tshuaj pom zoo kom maj mam yaj hauv qab tus nplaig. Cov kev txhaj tshwj xeeb ntawm cov tshuaj thiab lub sijhawm ntawm kev kho mob ncaj qha nyob ntawm tus neeg mob lub sijhawm. Yog li, cov ntaub ntawv no tau muab tshwj rau tus kws kho mob tom qab kuaj mob ntawm tus neeg mob.

Nws yuav tsum raug coj mus rau hauv tus account tias dextrose muaj peev xwm ua kom tsis muaj zog ntawm kev ua haujlwm ntawm cov mob glycosides vim ua tsis muaj zog thiab oxidation ntawm glycoside. Raws li, tsawg kawg ib teev yuav tsum elapse ntawm kev noj cov tshuaj no. Cov kua nplaum kuj txo cov kev ua haujlwm ntawm cov tshuaj zoo li no:

• nystatin
• analgesics
• streptomycin,
• adrenomimetic tshuaj.

Nrog kev ceev faj, cov tshuaj yog tawm rau hyponatremia thiab lub raum tsis ua haujlwm, tas li saib xyuas hauv central hemodynamics. Thaum lactation thiab thaum cev xeeb tub, kev siv ntawm dextrose yog qhia. Cov menyuam yaus hnub nyoog qis dua tsib xyoos tsis xaj tshuaj qabzib rau hauv daim ntawv ntawm cov ntsiav tshuaj, vim tias cov menyuam tsis paub yuav ua li cas noj cov tshuaj sublingually (nqus tau nyob hauv tus nplaig).

Yog tias cov ntshav qabzib ntau hauv tib neeg lub cev, hyperglycemia tuaj yeem txhim kho, cov yam ntxwv tseem ceeb ntawm kev tsis nqhis dej (polydipsia) thiab tso zis sai (polyuria). Nyob rau ntawm cov mob hnyav, mob sab laug ventricular tsis ua hauj lwm (ua tsis taus pa, hnoos, txog siav, mob ntsws ntsws) tshwm sim.

Cov tshuaj muaj nyob hauv ntau qhov ntau npaum:

• 0.5 mg ntsiav tshuaj
• 100 ml daws ntawm 10, 20 thiab 40 mg.

Cov piam thaj hauv cov ntsiav tshuaj yog xim dawb, tiaj-cylindrical thiab muaj kev pheej hmoo. Ib ntsiav tshuaj muaj 0.5 mg ntawm qhov yooj yim sib tov, dextrose monohydrate. Thiab tseem muaj tus lej ntawm cov khoom ntxiv: cov hmoov txhuv nplej siab, calcium stearate thiab talc. Daim ntawv teev cov ntsiav tshuaj ntawm cov tshuaj no tau sau tseg rau tus neeg mob txoj kev noj qab nyob zoo, txhim kho nws lub peev xwm ntawm lub cev thiab kev ua haujlwm hauv lub hlwb.

Piam thaj rau yog dab tsi?

Tib neeg lub cev xav tau cov piam thaj ua ib qho kev ntxim siab rau ntau yam tshuaj lom neeg. Tus txheej txheem no muaj nyob rau hauv qhov hloov chaw ntawm lub zog rau txhua lub hlwb ntawm lub cev thiab txuas ntxiv cov metabolism. Cov kua nplaum ua lub ntsej muag hauv lub zog, txhim kho txoj haujlwm ntawm cov qauv ntawm cellular. Thiab tseem muaj cov khoom no nkag rau hauv lub hlwb, saturates lawv nrog lub zog, txhawb kev cuam tshuam ntawm intracellular thiab pib cov txheej txheem ntawm kev hloov biochemical.

Kev tsis txaus ntawm monosaccharide nrog cov zaub mov ua rau malaise, muaj zog qaug zog thiab tsaug zog. Nrog kev tswj hwm cov tshuaj ntawm cov tshuaj tov nrog cov piam thaj, kev noj haus zoo ntxiv tshwm sim, cov nyhuv antitoxic zoo tuaj, thiab diuresis nce ntxiv. Nws tseem yuav tsum tau sau tseg ntawd piam thaj yog cov khoom tseem ceeb rau kev ua haujlwm ntawm lub siab nqaij.

Cov tshuaj no feem ntau siv hauv cov tshuaj rau kev kho mob ntawm ntau yam kab mob: lub hlwb tsis zoo, kab mob siab thiab lom. Ib qho tseem ceeb sib luag yog tias cov piam thaj yog qhov tsim nyog rau kev ua haujlwm ntawm lub hlwb. Nrog nws tsis muaj, teeb meem nrog concentration yog tau. Cov pa roj carbon monoxide no tseem tuaj yeem muaj qhov cuam tshuam ncaj qha rau lub xeev psychoemotional ntawm ib tus neeg, txhim kho thiab ua kom lub siab tus.

Cov tshuaj tseem raug pom zoo rau kev siv nyob rau hauv cov teebmeem hauv qab no:

1. Nrog hypoglycemia (ntshav tsis txaus ntshav qab zib).
2. Nrog rau lub cev qhuav dej (ntuav, plab zom mov).
3. Tom qab lom nrog hepatotropic tshuaj lom cov kab mob sib txawv.
4. Raws li cov ntshav hloov kua.

Cov tshuaj no feem ntau tau sau tseg rau cov hom phiaj prophylactic uas muaj cov vitamins tsis muaj zog, ua kom lub cev muaj zog, thaum lub sij hawm muaj kev loj hlob zoo lossis rov kho lub neej li qub tom qab muaj mob hnyav.

Cov lus qhia rau kev siv thiab ntau npaum li cas

Raws li cov lus qhia rau kev siv, qabzib hauv cov ntsiav tshuaj yog npaj rau kev siv sublingual, uas yog, resorption nyob rau hauv tus nplaig. Nws yuav tsum noj cov tshuaj kwv yees li ib teev thiab ib nrab ua ntej noj mov - qhov yuav tsum tau no yog vim qhov tseeb tias dextrose, uas yog ib feem ntawm cov tshuaj, txo cov qab los noj mov.

Qhov tsim nyog yuav tau cov tshuaj yog kws kho raws lub hnub nyoog thiab mob ntawm tus neeg mob:

• Thaum muaj tshuaj lom, 2-3 ntsiav tshuaj yog kws kho, siv sijhawm so ob teev,
• nrog cov mob ntshav qab zib mellitus, 1-2 ntsiav tshuaj tau qhia nrog lub sijhawm luv 5 feeb, muaj qhov mob me me ntawm tus kab mob, txog li 3 ntsiav tshuaj tau qhia nrog lub sijhawm nruab nrab ntawm ib nrab ib teev,
• rau cov menyuam yaus, kev coj ua txhua hnub (500 mg) tau muab faib ua ntau qhov tshuaj - txog 5 zaug hauv ib hnub, txog 3 xyoos, ntsiav tshuaj tsis tau sau ua sublingually - lawv yuav tsum tau diluted hauv dej.

Thaum cov piam thaj tau ua ke nrog ascorbic acid, yuav tsum tau saib xyuas lub raum, ntshav siab thiab cov tshuaj insulin kom tsim nyog.

Thaum cev xeeb tub thiab lactation

Hauv qee qhov xwm txheej yuav ntxiv cov tshuaj ascorbic acid nrog cov piam thaj nyob rau qee lub sijhawm. Feem ntau, cov tshuaj tau tawm rau lub cev tsis zoo. Thaum cev xeeb tub, lub hom phiaj tseem ceeb yog lub cev tsis txaus ntseeg hauv lub cev. Hauv lub hlis thib ob thiab thib peb, xav tau cov khoom no - tsawg kawg 90 mg ntawm cov piam thaj. Txawm li cas los xij, nws yog ib qho tseem ceeb uas yuav tsum xav txog tias kev noj ntau tshaj yuav ua rau muaj kev phom sij rau tus menyuam hauv plab. Tsis tas li ntawd, cov piam thaj tuaj yeem raug txiav txim thaum pub niam mis, tab sis qhov ntau tshaj ntawm cov tshuaj yog 120 mg.

Kev siv cov kua nplaum ntau dhau ua rau feem ntau ua rau cov xwm txheej hauv qab no:

1. Ua txhaum ntawm cov txheej txheem hauv metabolic.
2. Kev ua txhaum ntawm kev coj ua haujlwm ntawm cov txiav ua ntej thiab, yog li ntawd, teeb meem nrog kev tsim cov tshuaj insulin.
3. Ib qho kev nce siab hauv cov cholesterol thiab ntshav qab zib.
4. Qhov tsim ntawm cov ntshav txhaws thiab vascular plaques.
5. Cov tshuaj tiv thaiv tsis txaus ntawm tib neeg lub cev tsis txaus, ua los ntawm kev tsis haum.

Kev txuam nrog cov monosaccharide ntau dhau hauv cov ntshav muaj qhov tsis zoo rau cov hlab ntsha, uas ua rau lub cev tsis zoo ntawm txhua lub plab hnyuv siab raum. Yog li ntawd, tseem muaj peev xwm tsim cov kab mob atherosclerosis, mob raum, mob plawv, thiab txawm tias qhov muag tsis pom tseeb tsis tau txiav txim.

Cov xwm txheej hauv qab no yog kev cais tawm ntawm kev siv ntshav qabzib:

• mob raum tsis ua hauj lwm,
• lub plawv tsis ua haujlwm (hauv keeb kwm),
• nrog kev ntxhov siab hloov pauv sodium hauv cov ntshav,
• hyperhydration (muaj kua ntau dhau hauv lub cev),
• mob hlwb los sis mob ntsws ntsws,
• circulatory pathology.

Nws yuav tsum nco ntsoov tias rau cov menyuam yaus hnub nyoog qis dua 3 xyoos thiab rau cov neeg mob ntshav qab zib mellitus, cov tshuaj no tau sau nrog kev ceev faj thiab tsuas yog rau lub hom phiaj tshwj xeeb.

Mechanism ntawm kev ua

Glucose lossis dextrose yog qab zib uas yooj yim (monosaccharide). Lwm lub npe yog suab thaj qab zib. Nws yog ib feem ntawm cov suab thaj thiab cov carbohydrates: fructose, sucrose, hmoov txhuv nplej siab, maltose. Hauv cov txheej txheem ntawm kev ua kom lwj, cov carbohydrates nyuaj tau hloov pauv mus rau cov suab thaj yooj yim. Cov piam thaj hauv cov ntsiav tshuaj yog qhov yooj yim ntawm lub zog uas ua kom sai thiab ua kom tiav los ntawm lub cev. Dextrose yog koom nrog hauv cov txheej txheem hauv lub cev ntawm lub cev:

• txhawb txoj kev tsim thiab rhuav tshem ntawm cov roj ntsha,
• raws li cov txiaj ntsig ntawm cov kua nplaum ua, nucleoside triphosphate yog tsim, uas yog roj rau cov kabmob thiab cov nqaij hauv tib neeg lub cev,
• dextrose nourishes cov nqaij ntshiv thiab lub hlwb ntawm tus neeg.

Cov kua nplaum muaj nyob hauv lub hlwv ntawm 10 ntsiav tshuaj hauv ib qho. Cov Blisters muag ob qho tib si hauv 1 daim, thiab hauv thawv, ua 2 daim. 1 ntsiav tshuaj - 50 mg ntawm qabzib. Tus nqi nyob ntawm seb muaj pes tsawg leej ntsiav tshuaj thiab tshuaj noj. Tus nqi qis kawg rau ib lub ntsej muag yog los ntawm 6 rubles thiab siab dua.

Noj ntau dhau

Nrog kev noj ntau dhau ntawm cov tshuaj, hyperglycemia, ntshav siab ntxiv tshwm sim. Lub cev tsis tsim cov insulin ntau txaus uas tuaj yeem ua tiav cov kua nplaum uas tau txais. Hyperglycemia ua rau tsis muaj kev puas tsuaj rau cov hlab ntsha thiab cov plab hnyuv siab raum, thiab tseem ua rau kom lub xeev tsis xeev.

Nrog kev noj tshuaj ntau dhau ntawm cov piam thaj, mob taub hau, teeb meem ntawm lub plab zom mov, poob siab, pw tsaug zog muaj tshwm sim. Yog tias pom cov tsos mob tshwm sim, nws raug nquahu kom koj sab laj nrog kws kho mob los kho koj cov ntshav qab zib.

Mob ntshav qab zib ib txwm ua rau neeg tuag taus. Muaj ntshav qab zib ntau dhau yog qhov txaus ntshai heev.

Aronova S.M. muab cov lus qhia txog kev kho mob ntshav qab zib. Nyeem tag nrho

Nrog rau tus kab mob, mob ntshav qab zib

Mob ntshav qab zib, raws li cov lus qhia, yog ib qho ntawm contraindications rau kev noj dextrose hauv ntsiav tshuaj. Tab sis qee zaus tus kws kho mob tau sau ntawv rau cov tshuaj no rau cov neeg mob yog tias lawv muaj ntshav qab zib hom 1. Qhov no yog vim qhov tseeb tias cov neeg mob zoo li no tau qhia insulin hauv ntsiav tshuaj lossis lwm yam tshuaj muaj insulin. Thiab nrog lub ntsej muag ua kom lub ntsej muag nyob hauv lub cev (ntev ntev hauv cov zaub mov, muaj cov tshuaj insulin ntau, kev nyuaj siab lossis lub cev ntxhov siab, thiab lwm yam), cov thyroid hormone tsis tuaj yeem nkag mus rau hauv lub hlwb. Kev mob ntshav qab zib tsawg zuj zus, tshwm sim los ntawm kev tawm hws, tsis muaj zog, mob tachycardia, ua rau leeg huam. Qee zaum muaj kev tawm tsam sai sai.

Nws yog ib qho tseem ceeb kom nco ntsoov tias thaum tsis muaj kev pab tsim nyog, ib tus neeg uas muaj ntshav qab zib tuaj yeem poob rau qhov tsis nco qab. Txais tos ntawm qabzib sai sai normalizes qib ntawm cov piam thaj hauv cov ntshav, vim tias cov ntsiav tshuaj pib yuav tsum tau nqus tau thaum lub sijhawm resorption. Qhov tseem ceeb tshaj plaws tsis yog los cuam tshuam cov tsos mob ntawm hyperglycemia thiab hypoglycemia - lawv zoo sib xws. Yog tias muaj glucometer, ces koj yuav tsum ua kom ntsuas cov ntshav.

Nrog lub suab thaj txo qis hauv cov ntshav qab zib thiab tus neeg mob lub mob hnyav, cov kua nplaum yuav tsum raug noj txhua 5 feeb hauv qhov uas tus kws kho mob tau hais tseg.Lub teeb tawm tsam rau ntawm keeb kwm ntawm ntshav qab zib xav tau kev siv cov tshuaj txhua 20 feeb kom txog thaum tus neeg mob rov qab los. Cov lus qhia meej rau kev siv tshuaj yog muab los ntawm kws kho mob.

Nrog kev ua si khaus

Sau cov piam thaj thiab ncaws pob. Dextrose cov ntsiav tshuaj yog qhov xav tau thaum ua si kis las kom muaj qib ntawm cov piam thaj hauv ntshav, muab cov khoom noj carbohydrates rau cov leeg thiab lub siab.

Ua ntej kev cob qhia ntev ntev, kev ntaus pob ncaws pob coj tus nqi ntawm cov tshuaj pom zoo los ntawm cov kws tshwj xeeb. Qhov no yooj yim dua yog tias koj tsis tuaj yeem noj tag nrho hauv ib teev lossis ob teev ua ntej chav kawm. Cov kua nplaum muab lub zog uas koj xav tau rau kev qhia thiab tiv thaiv kev ua kom tsis muaj zog, kiv taub hau, thiab qaug zog heev tom qab kev tawm dag zog lub cev.

Thaum raug haus dej caw

Thaum lom nrog cawv thiab tshuaj muaj yees, cov hlwb ua rau muaj kev txom nyem. Txais tos cov qabzib tso cai koj ua kom muaj kev phom sij ua rau lawv, ua kom rov qab muab cov khoom noj muaj txiaj ntsig mus rau cov kab mob. Yog li, dextrose hauv cov ntsiav tshuaj muaj txiaj ntsig zoo rau kev kho mob ntawm cawv intoxication, kev quav yeeb tshuaj, tshem tawm los ntawm binge.

Tsis tas li ntawd, cov tshuaj hloov kho daim siab ua haujlwm, pab kom sai sai lub cev ntawm cov poov tshuaj lom toxins. Cov koob tshuaj uas tau pom zoo los ntawm tus kws tshaj lij hauv cov xwm txheej no yog noj txhua 2-3 teev.